by Dr. Tullio Simoncini
Extracted from Nexus Magazine,
Volume 14, Number 5 (August -
According to this
hypothesis based on years of scientific and clinical
research, the cause of cancer is infection by a common
The good news is that it can
with a powerful antifungal agent that can't
About the Author
Based in Rome, Italy, Dr Tullio Simoncini is a
medical doctor and surgeon specializing in oncology,
diabetology and metabolic disorders. He is also a Doctor
A humanitarian, he is opposed to any
kind of intellectual conformity, which he sees as often
based on suppositions without foundation or, worse, on
lies and falsehoods.
Dr Simoncini regularly
attends medical conferences and does interviews to
explain what's wrong with conventional cancer theories
and treatments, to present his fungal theory of cancer
and to describe case studies involving patients healed
with sodium bicarbonate, a powerful antifungal.
book, Cancer is a Fungus: A revolution in the therapy of
tumors (Edizioni Lampis), is available in Italian, Dutch
and English at his below website.
For more information on Dr Simoncini's theory, therapy
and case studies, and to view interviews and
testimonials, visit the portal website
My idea is that cancer doesn't depend on mysterious causes (genetic,
immunological or auto-immunological, as the official oncology
proposes), but it results from a simple fungal infection whose
destroying power in the deep tissues is actually underestimated.
The present work is based on the conviction, supported by many years
of observations, comparisons and experiences, that the necessary and
sufficient cause of the tumor is to be sought in the vast world of
the fungi, the most adaptable, aggressive and evolved
micro-organisms known in nature.
I have tried many times to explain this theory to leading
institutions involved in cancer issues (the Ministry of Health, the
Italian Medical Oncological Association, etc.), elaborating on my
thinking, but I have been brushed aside because of the impossibility
of setting my idea in a conventional context. A different,
international audience represents the possibility of sharing a view
about health which differs from what is widely accepted by today's
medical community, either officially or from the sidelines.
There is an opposition between the allopathic and the Hippocratic
medical ideal. The position that I promote represents instead a
meeting point of these two conceptions of health, since from the
conceptual point of view it sublimates and adds value to both, while
highlighting how they both are victims of a common conformist
The hypothesis of a fungal aetiology in chronic-degenerative
illness, able to connect the ethical qualities of the individual
with the development of specific pathologies, reconciles the two
orientations (allopathic and holistic) of medicine. The hypothesis
is a strong candidate for being that missing element of
psychosomatics that was sought but never found by one of the fathers
of psychosomatics, Viktor von Weiszacker.
In considering the biological dimensions of the fungi, for instance,
it is possible to compare the different degrees of pathogenicity in
relation to the condition of organs, tissues and cells of a guest
organism, which in turn also and especially depend on the behavior
of the individual.
Each time the recuperative abilities of a known psycho-physical
structure are exceeded, there is an inevitable exposure, even
considering possible accidental co-founders, to the aggression - even
at the smallest dimensions - of those external agents that otherwise
would be harmless.
In the presence of an indubitable connection
between patient morale and disease, it is no longer legitimate to
separate the two domains (allopathic and naturopathic) which are
both indispensable for improving the health of individuals.
mainstream theories on cancer causation
When facing the most pressing contemporary medical problem, cancer,
the first thing to do is to admit that we still do not know its real
cause. However treated in different ways by both official and
alternative medicine, cancer has an aura of mystery that still
exists around its real generative process.
The attempt to overcome the present impasse must therefore and
necessarily go through two separate phases: a critical one that
exposes the present limitations of oncology, and a constructive one
capable of proposing a therapeutic system based on a new theoretical
point of departure.
In agreement with the most recent formulation of
scientific philosophy, which suggests a counter-inductive approach
where it is impossible to find a solution with the conceptual tools
that are commonly accepted,1 only one logical formulation emerges:
to refuse the oncological principle which assumes that cancer is
generated by a cellular reproductive anomaly.
However, if the fundamental hypothesis of cellular reproductive
anomaly is questioned, it becomes clear that all the theories based
on this hypothesis are inevitably flawed.
It follows that both an auto-immunological process, in which the
body's defense mechanisms against external agents turn their
destructive capacity against internal constituents of the body, and
an anomaly of the genetic structure implicated in the development of
auto-destruction are inevitably disqualified.
Moreover, the common attempt to construct theories about multiple
causes that have an oncogenic effect on cellular reproduction
sometimes seems like a concealing screen, behind which there is
nothing but a wall. These theories propose endless causes that are
more or less associated with each other; and this means in reality
that no valid causes are found.
The invocation in turn of smoking,
alcohol, toxic substances, diet, stress, psychological factors,
etc., without a properly defined context, causes confusion and
resignation, and creates even more mystification around a disease
which may turn out to be simpler than it is depicted to be.
As background information, it is important to review the picture of
presumed genetic influences in the development of cancer processes
as they are depicted by molecular biologists. These are the
scientists who perform research on infinitesimally small cellular
mechanisms, but who in real life never see a patient. All present
medical systems are based on this research, and thus, unfortunately,
all therapies currently performed.
The main hypothesis of a genetic neoplastic causality is essentially
reduced to the fact that the structures and the mechanism in charge
of normal reproductive cellular activity become, for undefined
reasons, capable of an autonomous behavior that is disjointed from
the overall tissular economy.
The genes that normally have a
positive role in cellular reproduction are, then, imprecisely
referred to as "proto-oncogenes"; those that inhibit cellular
reproduction are called "suppressor genes" or "recessive oncogenes".
Both endogenous (never demonstrated) and exogenous cellular
factors - that is, those carcinogenic elements that are usually
invoked--are held responsible for the neoplastic degeneration of the
From a very superficial analysis of the presumed oncological
picture, however, it seems to be clear how the assertion of all this
unstoppable genetic hyperactivity can do nothing more that unveil
the abysmal stupidity that is at the basis of this way of conceiving
things. All those who work in the field do nothing but repeat the
stale litany of reproductive cellular anomalies on a genetic basis.
It is better to look for new horizons
and conceptual instruments that are capable of unearthing a real and
unique neoplastic aetiology.
In order to find the possible carcinogenic ens morbi on the horizon
of microbiology, it appears useful to return to the basic
taxonomical concepts of biology where we can see, incidentally, the
existence of a noticeable amount of indecision and indetermination.
Already in the last century, a German biologist, Ernst Haeckel
(1834-1919), departing from the Linnaeian concept that makes for two
great kingdoms of living things (vegetable and animal), denounced
the difficulties of categorizing all those microscopic organisms
which, because of their characteristics and properties, could not be
attributed to either the vegetable or the animal kingdom.
For these organisms, he proposed a third
kingdom, Protista (protists).
"This vast and complex world
includes a range of entities beginning with those that have
sub-cellular structure - existing at the limits of life - such as viroids and viruses, moving through the mycoplasms to, finally,
organisms of greater organization: bacteria, Actinomycetes,
Myxomycetes, fungi, protozoa and perhaps even some microscopic
The common element of these organisms is
the feeding system, which, being implemented (with very few
exceptions) by direct absorption of soluble organic compounds,
differentiates them both from animals and vegetables.
feed as above, but especially by ingesting solid organic materials
that are then transformed through the digestive process. Vegetables,
by utilizing mineral compounds and light energy, are capable of
feeding by synthesizing the organic substances.
The contemporary tendency of biologists is once again to pick up,
though in a more sophisticated way, the concept of the third
kingdom. One goes even further, however, arguing that within that
kingdom, fungi must be classified in a distinct category.
O. Verona3 says that if we put multicellular organisms provided with
photosynthetic capabilities (plants) in the first kingdom and the
organisms not provided with photosynthetic pigmentation (animals) in
the second kingdom - and organisms from both these kingdoms are made
of cells provided with a distinct nucleus (eukaryotes) - and,
furthermore, if we put in another kingdom (protists), those
monocellular organisms that have no chlorophyll and have cells that
are without a distinct nucleus (prokaryotes), the fungi can well
have their own kingdom because of the absence of photosynthetic
pigmentation, the ability to be monocellular and multicellular, and,
finally, their possession of a distinct nucleus.
Additionally, fungi possess a property that is strange when compared
to all other micro-organisms: the ability to have a basic
microscopic structure (hypha) with a simultaneous tendency to grow
to remarkable dimensions (up to several kilograms), keeping
unchanged the capacity to adapt and reproduce at any size.
From this point of view, therefore, fungi cannot be considered true
organisms, but cellular aggregates sui generis with an organismic
behavior, since each cell maintains its survival and reproductive
potential intact regardless of the structure in which it exists. It
is therefore clear how difficult it is to identify all the
biological processes in such complex living realities.
In fact, even today, there are huge
voids and taxonomical approximations in mycology.
It is worthwhile to examine more deeply this strange world, with
such peculiar characteristics, and try to highlight those elements
that somehow may be pertinent to the problems of oncology.
Fungi are heterotrophic
organisms and therefore need, as far as nitrogen and carbon
are concerned, pre-formed compounds. Of these compounds,
simple carbohydrates, for example monosaccharides (glucose,
fructose and mannose), are among the most utilized sugars.
This means that fungi, during their life cycle, depend on
other living beings which must be exploited in different
degrees for their feeding. This occurs both in a saprophytic
way (that is, by feeding on organic waste) and in a
parasitic way (that is, by attacking the tissue of the host
Fungi show a great variety of
reproductive manifestations (sexual, asexual, gemmation;
these manifestations can often be observed simultaneously in
the same mycete), combined with a great morphostructural
variety of organs. All of this is directed toward the end of
spore formation, to which the continuity and propagation of
the species is entrusted.
In mycology, it is often
possible to observe a particular phenomenon called
heterokaryon, characterised by the coexistence of normal and
mutant nuclei in cells that have undergone a hyphal fusion.
Nowadays, phytopathologists are quite worried about the
creation of individuals that are genetically quite different
even from the parents. This difference has taken place by
means of those reproductive cycles, which are called
parasexual. The indiscriminate use of phytopharmaceuticals
has in fact often determined mutations of the nuclei of many
parasitic fungi with the consequent creation of heterokaryon--and
this is sometimes particularly virulent in its
In the parasitic dimension,
fungi can develop from the hyphas more or less beak-shaped,
specialized structures that allow the penetration of the
The production of spores can be
so abundant as to include always, at every cycle, tens,
hundreds and even thousands of millions of elements that can
be dispersed at a remarkable distance from the point of
origin5 (a small movement is sufficient, for example, to
implement immediate diffusion).
Spores have an immense
resistance to external aggression, for they are capable of
staying dormant in adverse conditions for many years while
preserving unaltered their regenerative potentialities.
The development coefficient of
the hyphal apexes after the germination is extremely fast
(100 microns per minute under ideal conditions) with
ramification capacity, thus with the appearance of a new
apex region that in some cases is in the neighborhood of
The shape of the fungus is never
defined, for it is imposed by the environment in which the
fungus develops. It is possible to observe, for example, the
same mycelium in the simple isolated hyphas status in a
liquid environment or in the form of aggregates that are
increasingly solid and compact, up to the formation of
pseudoparenchymas and of filaments and mycelial strings.7
By the same token, it is
possible to observe in different fungi the same shape
whenever they must adapt to the same environment (this is
called dimorphism). The partial or total substitution of
nourishing substances induces frequent mutations in fungi,
and this is further proof of their high adaptability to any
When the nutritional conditions
are precarious, many fungi react with hyphal fusion (among
nearby fungi) which allows them to explore the available
material more easily, using more complete physiological
processes. This property, which substitutes co-operation for
competition, makes them distinct from any other
micro-organism, and for this reason Buller calls them social
When a cell gets old or becomes
damaged (e.g., by a toxic substance or by a pharmaceutical),
many fungi whose intercellular septums are provided with a
pore react by implementing a defense process called
protoplasmic flux, through which they transfer the nucleus
and cytoplasm of the damaged cell into a healthy one, thus
conserving unaltered all their biological potential.
The phenomena regulating the
development of hyphal ramification are unknown to date.9
They consist of either a rhythmic development or in the
appearance of sectors which, though they originate from the
hyphal system, are self-regulating,10 that is, independent
of the regulating action and behavior of the rest of the
Fungi are capable of
implementing an infinite number of modifications to their
own metabolism in order to overcome the defense mechanism of
the host. These modifications are implemented through
plasmatic and biochemical actions as well as by a volumetric
increase (hypertrophy) and numerical hyperplasy of the cells
that have been attacked.11
Fungi are so aggressive as to
attack not only plants, animal tissue, food supplies and
other fungi, but even protozoa, amoebas and nematodes.
Fungi hunt nematodes, for example, with
peculiar hyphal modifications that constitute real mycelial criss-cross,
viscose or ring traps that immobilize the worms.
In some cases, the aggressive power of the fungus is so great as to
allow it - with only a cellular ring made up of three unit - to
tighten its grip, capture and kill its prey within a short time,
notwithstanding the desperate struggling of the prey.
From the short notations above, it therefore seems fair to dedicate
greater attention to the world of fungi, especially considering the
fact that biologists and microbiologists constantly highlight large
deficiencies and voids in all their descriptions and interpretations
of fungi's shapes, physiologies and reproductions.
So the fungus, which is the most powerful and the most organized
micro-organism known, seems to be an extremely logical candidate as
a cause of neoplastic proliferation.
Imperfect fungi (so called because of the lack of knowledge and
understanding of their biological processes) deserve particular
attention, since their essential prerogative sits in their
The greatest disease of mankind may therefore hide within a small
cluster of pathogenic fungi, and may after all be located with just
some simple deductions able to close the circle and provide the
albicans - a necessary and sufficient cause of cancer
Considering that among the human parasite species the Dermatophytes
and Sporotrichum demonstrate an excessively specific morbidity, and
that experience shows that Actinomycetes, Toluropsis and Histoplasma
rarely enter the context of pathology, the Candida albicans fungus
clearly emerges as the sole candidate for tumor proliferation.
If we stop for a second and reflect on its characteristics, we can
observe many analogies with neoplastic disease.
The most evident are:
1) ubiquitous attachment - no
organ or tissue is spared
2) the constant absence of hyperpyrexia
3) sporadic and indirect involvement of the differential
4) invasiveness that is almost exclusively of the focal type
5) progressive debilitation
6) refractivity to any type of treatment
7) proliferation facilitated by multiplicity of indifferent
8) symptomatological basic configuration with structure
tending to the chronic
Therefore, an exceptionally high and
diversified pathogenic potentiality exists in this mycete of just a
few microns in size, which, even though it cannot be traced with the
present experimental instruments, cannot be neglected from the
clinical point of view.
Certainly, its present nosological classification cannot be
satisfactory because, if we do not keep the possibly endless
parasitic configurations in mind, that classification is too
simplistic and constraining.
We therefore have to hypothesize that Candida, in the moment it is
attacked by the immunological system of the host or by a
conventional antimycotic treatment, does not react in the usual,
predicted way but defends itself by transforming itself into
ever-smaller and non-differentiated elements that maintain their
fecundity intact to the point of hiding their presence both to the
host organism and to possible diagnostic investigations.
Candida's behavior may be considered to be almost elastic. When
favorable conditions exist, Candida thrives on an epithelium; as
soon as the tissue reaction is engaged, it massively transforms
itself into a form that is less productive but impervious to attack:
the spore. If, then, continuous subepithelial solutions take place,
coupled with a greater areactivity in that very moment, the spore
gets deeper into the lower connective tissue in such an impervious
state that colonization is irreversible.
In fact, Candida takes advantage of a structural interchangeability,
utilizing it according to the difficulties, e.g., in feeding, to
overcome its biological niche. In this way, Candida is free to
expand to maturation in the soil, air, water, vegetation, etc. - that
is, wherever there is no antibody reaction.
In the epithelium,
instead, it takes a mixed form, which is reduced to the sole spore
component when it penetrates the lower epithelial levels, where it
tends to expand again in the presence of conditions of tissular
The initial mandatory step of an in-depth research endeavor would be
to understand if and in which dimensions the spore transcends, what
mechanisms it engages to hide itself or, again, to preserve its
parasitic characteristic, or if it has available a neutral quiescent
position which is difficult or even impossible to detect by the
Unfortunately, today we do not have the appropriate means, either
theoretical or technical, to answer these and similar questions, so
the only valid suggestions can come solely from clinical observation
and experience. While not providing immediate solutions, these
sources can at least stimulate further questions.
Assuming that Candida albicans is the
agent responsible for tumor development, a targeted therapy would
take into account not just its static and macroscopic manifestations
but even the ultramicroscopic ones, especially in their dynamic
valency, that is, the reproductive. It is very probable that the
targets to attack are the fungi's dimensional transition points in
order to perform a decontamination with such a scope as to include
the whole spectrum of the biological expression--parasitic,
vegetative, sporal and even ultradimensional and, to the limit,
If we stop at the most evident phenomena, we risk administering
salves and unguents forever (in the case of dermatomycosis or in
psoriasis), or clumsily attacking (with surgery, radiotherapy or
chemotherapy) enigmatic tumoural masses with the sole result of
facilitating their propagation, which is already heightened in the
Why, one may ask, should we assume a different and heightened
activity of Candida albicans, since it has been abundantly described
in its pathological manifestations? The answer lies in the fact that
it has been studied only in a pathogenic context, that is, only in
relation to the epithelial tissues.
In reality, Candida possesses an aggressive valency that is
diversified in function in the target tissue. It is just in the
connective or in the connective environment, in fact, and not in the
differentiated tissues, that Candida may find conditions
to an unlimited expansion. This emerges if we stop and reflect for a
moment on the main function of connective tissue, which is to convey
and supply nourishing substances to the cells of the whole organism.
This is to be considered as an environment external to the more
differentiated cells such as nervous, muscular, etc. It is in this
context, in fact, that the alimentary competition takes place.
On the one hand, we have the organism's cellular elements trying to
defeat all forms of invasion; on the other hand, we have fungal
cells trying to absorb ever-growing quantities of nourishing
substances, for they have to obey the species' biological imperative
to form ever larger and diffused masses and colonies.
From the combination of various factors
pertinent to both the host and the aggressor, it is possible to
hypothesize the evolution of a candidosis.
First stage: Integer epitheliums,
absence of the debilitating factors. Candida can only exist as a
Second stage: Non-integer
epitheliums (erosions, abrasions, etc.), absence of stage
debilitating factors, unusual transitory conditions (acidosis,
metabolic disorder, and microbial disorder). Candida expands
superficially (classic mycosis, both exogenous and endogenous).
Third stage: Non-integer
epitheliums, presence of debilitating factors (toxic, stage
radiant, traumatic, neuropsychic, etc.). Candida goes deeper
into the subepithelial levels, from which it can be carried to
the whole organism through the blood and lymph (intimate
Stages one and two are the most studied
and understood, while stage three, though it has been described in
its morphological diversity, is reduced to a silent form of
This is not acceptable from a logical point of view,
because no one can demonstrate the harmlessness of the fungal cells
in the deepest parts of the organism.
In fact, the assumption that Candida can behave in the same
saprophytic manner that is observed on integer epitheliums when it
has successfully penetrated the lower levels is at least risky,
because the assumption would have to be sustained by concepts that
are totally aleatory (i.e., dependent on chance).
In fact, we are asked not only to accept a priori that the
connective environment is,
not suitable to nourish the
Candida, but also at the same time to accept
the omnipotence of the body's
defense system towards an organic structure that is invasive
but that then becomes vulnerable once lodged in the deeper
As for point (a), it is difficult to
imagine that a micro-organism so able to adapt itself to any
substrata cannot find elements to support itself in the human
organic substance; by the same token, it seems risky to hypothesize
that the human organism's defense system is totally efficient at
every moment of its existence.
As for point (b), the assumption that
there is a tendency to a state of quiescence and vulnerability in
the case of a pathogenic agent such as fungus--the most invasive and
aggressive micro-organism existing in nature - seems to carry a whiff
of the irresponsible.
It is therefore urgent, on the basis of the abovementioned
considerations, to recognize the hazardous nature of such a
pathogenic agent which is capable of easily taking the most various
biological configurations, both biochemical and structural,
regardless of the conditions of the host organism.
The fungal expansion gradient in fact becomes steeper as the tissue
that is the host of the mycotic invasion becomes less eutrophic and
thus less reactive.
To that end, it seems useful to consider briefly the "benign tumor"
This is an issue that always appears in general
pathology but is brushed aside most of the time too easily, and it
is overlooked because it usually doesn't create either problems or
worries. It constitutes one of those underestimated grey areas
seldom subjected to rational, fresh consideration.
If the benign tumor, however, is not considered a fully fledged
tumor, it would be advantageous, for clarity, to categorize it in an
appropriate nosological scheme.
If it is thought that, instead, it fully belongs to neoplastic
pathology, then it is necessary to consider its non-invasive
character and consequently to consider the reasons for this.
It is in fact evident how in this second scenario, the thesis based
on a presumed predisposition of the organism to auto-phagocytosis,
having to admit an expressive graduation, would stumble into such
additional difficulties such as to become extremely improbable.
By contrast, in the fungal scenario, the mystery of why there are
benign and malignant tumors is exhaustively solved, since they can
be recognized as having the same aetiological genesis.
The benignity or malignancy of a cancer in fact depends on the
capability of tissular reaction of a specific organ expressing
itself ultimately in the ability to encyst fungal cells and to
prevent them from developing in ever-larger colonies. This can be
achieved more easily where the ratio between differentiated cells
and connective tissue is in favor of the former.
Situated between the impervious noble tissues, then, and the
defenseless connective tissues, the differentiated connective
structures (the glandular structures in particular) represent that
medium term which is only somewhat vulnerable to attack because of
an ability to offer a certain type of defense.
And it is in these conditions that benign tumors are formed; that
is, where the glandular connective tissue is successful in forming
hypertrophic and hyperplastic cellular embankments against the
parasites. In the stomach and in the lung, instead, since there are
no specific glandular units, the target organ, provided with a small
defensive capability, is at the mercy of the invader.
Furthermore, it is worth mentioning how several types of intimate
fungal invasion do not determine the appearance of malignant or
benign tumors but a type of particular benign tumor (specific
degenerative alterations), as is the case with some organs or
apparatuses that do not have peculiar glandular structures but
nevertheless are attacked in their connective tissue, although in a
In fact, if we consider multiple sclerosis, SLA, psoriasis, nodular
panarthritis, etc., the possible development of the fungus in a
three-dimensional sense is actually limited by the anatomic
configuration of the invaded tissues, so that only a longitudinal
expansion is allowed.
Going back to the precondition of areactivity that is necessary for
neoplastic development in a specific individual, it is permissible
to affirm how in the human body each external or internal element
that determines a reduction of well-being in an organism, organ or
tissue possesses oncogenic potentiality. This is not so much because
of an intrinsic damaging capability as much as a generic property of
favoring the fungal (that is, tumoral) flourishing.
Then the causal network so much invoked in contemporary oncology,
which involves toxic, genetic, immunological, psychological,
geographical, moral, social and other factors, finds a correct
classification only in a mycotic infectious perspective where the
arithmetical and diachronic summation of harmful elements works as a
co-factor to the external aggression.
treatments vs. antifungal therapy
With the theoretical basis of the tumor/fungus equivalency
demonstrated, it is clear how this interpretative key offers a long
series of questions concerning contemporary therapies, both
oncological (used without reference indexes) and antimycotic (utilized
only at a superficial level).
Which path is best to walk today, then, when faced with a cancer
patient, since the conventional oncological treatment, not being
aetiological, can only occasionally have positive effects and most
of the time produces damage?
In the fungal perspective, in fact, the effectiveness of surgery is
noticeably reduced because of the extreme diffusibility and
invasiveness characteristic of a mycelial conglomerate. Surgery to
solve the problem is therefore tied to the case; that is, to
conditions in which one has the luck to be able to remove the entire
colony completely (which is often possible in the presence of a
sufficient encystment, but only where benign tumors are concerned).
Chemotherapy and radiotherapy produce almost exclusively negative
effects, both for their specific ineffectiveness and for their high
toxicity and harmfulness to the tissues, which in the last analysis
favors mycotic aggressiveness.
By contrast, an antifungal, antitumor-specific therapy would take
into account the importance of the connective tissue together with
the reproductive complexity of fungi. Only by attacking the fungi
across the spectrum of all its forms, at points where it is most
vulnerable from the nutritional point of view, would it be possible
to hope to eradicate them from the human organism.
The first step to take, therefore, would be to reinforce the cancer
patient with generic reconstituent measures (nutrition, tonics,
regulation of rhythms and vital functions) that are able to enhance
the general defenses of the organism.
Concerning the possibility of having available pharmaceutical cures,
which unfortunately do not exist today, it seems useful, in the
attempt to find an antifungal substance that is quite diffusible and
therefore effective, to consider the extreme sensitivity of Candida
towards sodium bicarbonate (i.e., in the oral candidosis of
breastfed babies). This is consistent with the fact that Candida has
an accentuated ability to reproduce in an acid environment.
Theoretically, therefore, if treatments could be found that put the
fungus in direct contact with high sodium bicarbonate (NaHCO3)
concentrations, we should be able to see a regression of the tumoral
And this is what happens in many types of tumor, such as colon and
liver - and especially stomach and lung, the former susceptible to
regression just because of its "external" anatomic position, and the
latter because of the high diffusibility of sodium bicarbonate in
the bronchial system and for its high responsiveness to general
By applying a similar therapeutic approach, it has been possible in
many patients to achieve complete remission of the symptomatology
and normalization of the instrumental data.
It is important to emphasize that these cases are just an example of
what could be a new way of perceiving the complexity of medical
problems, especially in oncology.
[Reports of seven cases of patients, several of whom have been
documented for 10 years following sodium bicarbonate treatment, are
summarized in the complete article at the web page
It seems appropriate to analyze, in a critical and self-critical
spirit, what may emerge in neoplastic pathology that is new and
If we closely observe the proposed therapeutic approach,
it is possible to see that, independently of its real effectiveness,
it has value as an innovative theory. First, it challenges the
present methodology and especially its assumptions. Second, it
offers a concrete alternative proposal to a mountain of conjectures
and postures that sound authoritative but are too generic and
The identification of one tumoral cause, even with all the possible
general provisos, would represent a step forward that is
indispensable for escaping that passivity determined by a lack of
results, and which is responsible for medical behaviors that are
based too much on faith and not enough on real confidence.
Given, therefore, that an unconventional medical approach can
benefit some patients better from any point of view than the
official treatments, and since valuable results can be demonstrated,
this should stimulate us to pursue further research while avoiding
patronizing postures that are both limiting and non-productive.
We can therefore discuss whether or not sodium bicarbonate is the
real reason for the recoveries or if, instead, those recoveries are
due to the interaction of a number of conditions that have been
created, the results of unidentified neuropsychical factors, or
maybe the results of something totally unknown. What is beyond
question, however, is the fact that a certain number of people, by
not following conventional methods, have been able to go back to
normality without suffering and without mutilation.
The message of this experience is therefore a call to search for
those solutions that are in accord with the simple Hippocratic
obligation to man's "well-being"; that is, we must be stimulated to
a critical evaluation of our contemporary oncological therapies
which indubitably can guarantee suffering.
When we group together
both malignant tumors that are occasionally or never healed (such as
lung and stomach) and tumors that border with benignity (such as the
majority of thyroid and prostatic tumors, etc.) or put them together
with those that have an autonomous positive outcome notwithstanding
chemotherapy (i.e., infantile leukaemia) - all of this appears to be
devious and misleading, having only the purpose of forging a
consensus that would otherwise be impossible to obtain with
intellectually ethical behavior.
The fact that modern medicine not only cannot offer sufficient
interpretative criteria but even uses dangerous methodologies that
are also harmful and meaningless-even if carried out with good faith
is something which must push us all to search for humane and logical
alternatives. At the same time, it is necessary to carefully,
open-mindedly and logically consider any theory or point of view
that is dared to be advanced in the battle against that monstrous
and inhuman yoke that is the tumor.
To this end, a note of acknowledgement is to go to all those who are
aware of the harmfulness of conventional therapeutic methods and
constantly try to find alternative solutions.
People like Di Bella,
Govallo and others, although guilty of utilizing the same
inauspicious principles of official medicine (thus showing an
excessively conformist mindset), are actually using common sense by
trying to relieve the suffering of cancer patients through the use
of painless methodologies, and in some cases are able to achieve
remissions, even though they're in the dark about the real causes of
In an alternative perspective, then, it would be necessary to
conceive a new approach to experimentation in the oncological field,
setting epidemiological, aetiological, pathogenic, clinical and
therapeutic research in line with a renewed microbiology and
mycology that would probably drive us to the conclusion already
illustrated: that is, the tumor is a fungus
The possible discovery that not only tumors but also the majority of
chronic degenerative disease could be reconciled to mycotic
causality would represent a qualitative quantum leap, which, by
revolutionizing medical thinking, could greatly improve life
expectancy and quality of life.
Such reconciliation might include a
wider spectrum of fungal parasites (for example, in diseases of the
connective tissues, multiple sclerosis, psoriasis, some epileptic
forms, diabetes type 2, etc.).
In closing, considering that the world
of fungi-those most complex and aggressive micro-organisms-has been
bypassed and left unobserved for far too long, the hope of this work
is to promote awareness of the hazards of these micro-organisms so
that medical resources can be channeled not up blind alleys but
towards the real enemies of the human organism: external infectious
Addendum - A
Note on Cancer Treatment
The implications from my hypothesis that cancer is a fungus which
can be eradicated with
sodium bicarbonate are that:
eighty years of genetic study
and application has been for nothing, especially considering
that the genetic theory of cancer has never been
the loss of millions, if not
billions, of lives with all the suffering has been for
the billions of dollars spent on
chemotherapy medicine, radiotherapy, etc. has been for
the recognition and prizes given
to eminent researchers and professors has been for nothing
the oncologist could be replaced
by the family doctor
the pharmaceutical industry will
incur tremendous financial losses (sodium bicarbonate is
inexpensive and impossible to patent)
My methods have cured people for 20
years. Many of my patients recovered completely from cancer, even in
cases where official oncology had given up.
The best way to try to eliminate a tumor is to bring it into
contact with sodium bicarbonate, as closely as possible, i.e., using
oral administration for the digestive tract, an enema for the
rectum, douching for the vagina and uterus, intravenous injection
for the lung and the brain, and inhalation for the upper airways.
Breasts, lymph nodes and subcutaneous
lumps can be treated with local perfusions. The internal organs can
be treated with sodium bicarbonate by locating suitable catheters in
the arteries (of the liver, pancreas, prostate and limbs) or in the
cavities (of the pleura or peritoneum). (Note that sodium
bicarbonate should not be used as a cancer preventive.)
It is important to treat each type of cancer with the right dosage.
For phleboclysis (drip infusion), 500 cc given in a series of
intervals - 5% strength on one day and 8.4% the next - is required,
depending on the patient's weight and condition; the stronger dose
may perhaps be needed in cases of lung and brain cancers according
to the tumor type (primary or metastatic) and size. For external
administrations, it is enough to taste if the solution is salty.
Sometimes it is judicious to combine different administrations.
For each treatment, take into consideration that tumor colonies
regress between the third and fourth day and collapse between the
fourth and fifth, so a six-day administration is sufficient. A
complete, effective cycle is made up of six treatment days on and
six days off, repeated four times. The most important side effects
of this care system are thirst and weakness.
For skin cancers (melanoma, epithelioma, etc.), a 7% iodine tincture
should be spread on the affected area once a day, 20-30 times
consecutively in one sitting, with the aim of producing a number of
layers of crust.
If, after one month of treatment, the first crust
is gone and the skin is not completely healed, then the treatment
should be continued in the same manner until the second crust forms,
heals and then comes loose without any assistance. (The procedure is
also applicable for treating psoriasis.)
After this treatment, the
cancer will be gone and stay away forever.
For more information, see "Protocol Treatments with sodium
biocarbonate solutions" at
and FAQ sections at
Due to space constraints, we are unable to reprint Dr Simoncini's
paper in full. To download the complete paper including case study
summaries, go to the web page
1. Feyerabend, P.K., Contro il
metodo ("Against Method"), Feltrinelli, Milano, 1994, p. 26
2. Verona, O., Il vasto mondo dei funghi ("The Vast World of
Fungi"), Edizioni Nuova Italia, Firenze, 1973, p. 1
3. op. cit., p. 2
4. Rambelli, A., Fondamenti di micologia ("Basics of Mycology"),
Edizioni Guida, Napoli, 1972, p. 35
5. op. cit.
6. op. cit., p. 28
7. Verona, op. cit., p. 5
8. Rambelli, A., op. cit., p. 31
9. op. cit., p. 28
10. op. cit., p. 29
11. op. cit., p. 266
12. op. cit., p. 273